Download Why We Get Fat: And What to Do About It (Borzoi Books) [Deckle Edge] [Hardcover] .pdf

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2 Responses to “Download Why We Get Fat: And What to Do About It (Borzoi Books) [Deckle Edge] [Hardcover] .pdf”

  • maramaye "maramaye" says:
    900 of 970 people found the following review helpful:
    5.0 out of 5 stars
    Not another “balanced eating and exercise” book, December 28, 2010
    By 
    maramaye “maramaye” (Seattle, WA United States) –
    Amazon Verified Purchase(What’s this?)

    The brilliant thing about science is that when something is disproved once, it’s disproved forever. The not-so-brilliant thing about public health policy is that it has little to do with science.

    Everyone in the developed world knows what’s causing our obesity epidemic. BBC nailed it: “We eat too much, and too much of the wrong things,” and Michelle Obama tells us “We have to move more.” Clearly what we need is a balanced diet of lean meats, some good fats, and complex carbohydrates like fruit, vegetables and whole grain bread, and exercise of 30 to 90 minutes per day. Their prescription is completely reasonable and makes intuitive sense.

    It is neat, plausible, and wrong. It has in fact been disproved, as nearly as “disproof” can exist in nutrition science.

    In his previous book, Good Calories Bad Calories, respected science journalist Gary Taubes exhaustively researched and cited two centuries worth of research in nutrition. He came to the conclusion that none of those recommendations is supported by science, because the fundamental theory on which they’re based is wrong. Why We Get Fat is an updated summary of that earlier work, much quicker and easier to read, with some significant points clarified.

    The most important point of the book is that all those public recommendations — the food pyramid, the “eat food, not too much” approach, everything we know about a balanced lifestyle — is founded on the premise of Calories In vs. Calories Out. That we get fat because we eat too many calories, or we don’t burn enough of them through movement. But this is nonsense. It’s not just wrong, it is actually not a statement about what causes obesity at all (or heart disease, cancer or diabetes, for that matter.) It is, in Taubes’ words, a “junior high level mistake,” because it tells us nothing about fat accumulation. If we get fat, by definition we have taken in more calories than we’ve put out — but WHY we took in those calories, or didn’t burn them, is the key point.

    Taubes reviews the scientific literature (rather than the popular press) and presents a conclusion that was common knowledge before WWII, and heresy afterward: we get fat because our fat cells have become disregulated and are taking nutrients that should be available to other tissues. Like a tumor, the cells live for themselves rather than in balance with the rest of the body. And since those nutrients aren’t available, we become hungry and tired. Therefore we eat more, and move less.

    For the chronic dieters among us, one passage about animal models will explain decades of frustration. Rodents with a particular part of the hypothalamus destroyed would become obese and/or sedentary *as a consequence* of their bodies putting on more fat. “After the surgery, their fat tissue sucks up calories to make more fat; this leaves insufficient fuel for the rest of the body…The only way to prevent these animals from getting obese is to starve them…they get fat not by overeating but by eating at all.” Sound familiar?

    The problem isn’t one of gluttony and sloth, as Taubes refers to it, but of hormone balance. Simply put, some people are more sensitive to the hormone effects of insulin, cortisol, and a few other -ols, than other people are. The more sensitive you are, the more you’re likely to get fat, and the more fat you’re likely to get, in the presence of even small amounts of carbohydrate — and in the absence of enough fat.

    That’s right, this book advocates eating fat. Not just moderately, but as much fat as possible, up to 78% of calories. Not lean meats, not Jenny-O 99.6% fat-free turkey, not skinless chicken breasts, but lard. Yes, lard. The healthy way of eating, according to Taubes, is moderately high protein and high fat. Yes, high fat. About a 3:1 ratio of fat to protein, and almost no carbohydrates. (Telling people to eat a balanced diet containing carbohydrates is, he says, equivalent to telling smokers to include a balanced serving of cigarettes.) And he demonstrates exactly why a high-fat, low-carbohydrate diet is the most heart-healthy approach, as borne out by several dozen recent studies.

    While Taubes acknowledges that exercise seems to be good for us for a variety of reasons, weight control isn’t one of them. Study after study conducted by proponents of exercise have admitted that they see no compelling evidence for exercise as a weight-loss tool. And it makes sense if you throw out the calories in/calories out model of why we get fat. If we’re fat because our fat tissues are starving the rest of our cells of fuel, exercise is just going to make us hungrier and more tired, not leaner and more fit. (It’s worth noting that according to Taubes, in the 1930s obese patients were treated with bed rest.)

    [This review was edited to clarify the following point.] The main thrust of Taubes’ argument, however, surrounds…

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  • Steven Parker "M.D." says:
    327 of 378 people found the following review helpful:
    5.0 out of 5 stars
    Hie Thee Away, Farinaceous Foods and Saccharine Matter!, December 28, 2010
    By 
    Steven Parker “M.D.” (Arizona USA) –

    At the start of my medical career over two decades ago, many of my overweight patients were convinced they had a hormone problem causing it. I carefully explained that’s rarely the case. As it turns out, I may have been wrong. And the hormone is insulin.

    Mr. Taubes wrote this long-awaited book for two reasons: 1) to make the ideas in his 2007 tour de force Good Calories, Bad Calories: Fats, Carbs, and the Controversial Science of Diet and Health (Vintage) more accessible to the public, and 2) to speed up the process of changing conventional wisdom on overweight. GCBC was the equivalent of a college-level science course. Many nutrition science geeks loved it while recognizing it was too difficult for the average person to digest.

    The author hopes to convince us that “We don’t get fat because we overeat; we overeat because we’re getting fat.” We need to think of obesity as a disorder of excess fat accumulation, then ask why the fat tissue isn’t regulated properly. A limited number of hormones and enzymes regulate fat storage; what’s the problem with them?

    Mr. Taubes rejects the old “calories-in/calories-out” theory of fat accumulation. You remember the equation: eat too many calories and you get fat, or fail to burn up enough calories with metabolism and exercise, and you get fat. To lose fat, eat less and exercise more. [He admits it has at least a little validity.] We’ve operated under that theory for the last half century, but keep getting fatter and fatter. So the theory must be wrong on the face of it. Is there a better one?

    Here is Taubes’s. The hormone in charge of fat strorage is insulin; it works to make us fatter, building fat tissue. If you’ve got too much fat, you must have too much insulin action. And what drives insulin secretion from your pancreas? Dietary carbohydrates, especially refined carbs such as sugars, flour, cereal grains, starchy vegetables (e.g., corn, beans, rice, potatoes), liquid carbs. These are the “fattening carbs.” Dozens of enzymes and hormones are at play either depositing fat into tissue, or mobilizing the fat to be used as energy. Any regulatory derangement that favors fat accumulation will CAUSE gluttony (overeating) or sloth (inactivity). So it’s not your fault. Cut back on carb consumption to lower your fat-producing insulin levels, and you turn fat accumulation into fat mobilization.

    Before you write off Taubes as a fly-by-night crackpot, note that he’s received three Science-in-Society Journalism Awards from the National Association of Science Writers. He’s a respected, professional science writer.

    Heres an example to illustrate how hormones control growth of tissues. Consider the transformation of a skinny 11-year-old girl into a voluptuous woman of 18. Various hormones make her grow and accumulate fat and other tissues in the places we now see curves. The hormones make her eat more, and they control the final product. The girl has no choice. Same with our adult fat tissue, but with a different mix of hormones.

    Taubes’s writing is clear and persuasive. He lays out a logical series of facts and potential connections and explanations, leading you to see things his way. If insulin controls fat storage by building and maintaining fat tissue, and if carboydrates drive insulin secretion, then the way to reduce overweight and obesity is carbohydrate-restricted eating, especially avoiding the fattening carbohydrates. I’m sure that’s true for many folks, perhaps even a majority.

    At the back of the book is a simple, university-affiliated low-carb eating plan.

    Controlling excess body fat by avoiding carbohydrates was the conventional wisdom prior to 1960, as documented by Mr. Taubes. Low-carb diets for obesity date back almost 200 years.

    Mr. Taubes provides numerous scientific references to back his assertions. I checked out one in particular that didn’t sound right. Some background first. Reducing our total fat and saturated fat consumption over the last 40 years was supposed to lower our LDL cholesterol, thereby reducing the burden of coronary heart disease, which causes heart attacks. Instead, we’ve experienced the obesity epidemic as those fats were replaced by carbohydrates. Taubes mentions a 2009 medical journal article by Kuklina, implying that Kuklina says the number of heart attacks has not decreased as we’ve made these diet changes. Kuklina doesn’t say that. In fact, age-standardized heart attack rates have decreased in the U.S. during the last decade.

    Furthermore, autopsy data document a reduced prevalence of anatomic coronary heart disease in people aged 20-59 from 1979 to 1994, but no change in prevalence for those over 60. The incidence of coronary heart disease decreased in the U.S. from 1971 to 1998 (the…

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